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Coral Reef Socio-Ecological Systems Analysis & Restoration

Restoration strategies for coral reefs are usually focused on the recovery of bio-physical characteristics. They seldom include an evaluation of the recovery of the socio-ecological and ecosystem services features of coral reef systems. This paper proposes a conceptual framework to address both the socio-ecological system features of coral reefs with the implementation of restoration activity for degraded coral reefs. Such a framework can lead to better societal outcomes from restoration activities while restoring bio-physical, social and ecosystem service features of such systems. We first developed a Socio Ecological System Analysis Framework, which combines the Ostrom Framework for analyzing socio-ecological systems and the Kittinger et al. human dimensions framework of coral reefs socio-ecological systems. We then constructed a Restoration of Coral Reef Framework, based on the most used and recent available coral reef restoration literature. These two frameworks were combined to present a Socio-Ecological Systems & Restoration Coral Reef Framework. These three frameworks can be used as a guide for managers, researchers and decision makers to analyze the needs of coral reef restoration in a way that addresses both socio-economic and ecological objectives to analyze, design, implement and monitor reef restoration programs.

2018

Copper Oxide Nanoparticles Stimulate the Immune Response and Decrease Antioxidant Defense in Mice After Six-Week Inhalation

Copper oxide nanoparticles (CuO NPs) are increasingly used in various industry sectors. Moreover, medical application of CuO NPs as antimicrobials also contributes to human exposure. Their toxicity, including toxicity to the immune system and blood, raises concerns, while information on their immunotoxicity is still very limited. The aim of our work was to evaluate the effects of CuO NPs (number concentration 1.40×106 particles/cm3, geometric mean diameter 20.4 nm) on immune/inflammatory response and antioxidant defense in mice exposed to 32.5 µg CuO/m3 continuously for 6 weeks. After six weeks of CuO NP inhalation, the content of copper in lungs and liver was significantly increased, while in kidneys, spleen, brain, and blood it was similar in exposed and control mice. Inhalation of CuO NPs caused a significant increase in proliferative response of T-lymphocytes after mitogenic stimulation and basal proliferative activity of splenocytes. CuO NPs significantly induced the production of IL-12p70, Th1-cytokine IFN-γ and Th2-cytokines IL-4, IL-5. Levels of TNF-α and IL-6 remained unchanged. Immune assays showed significantly suppressed phagocytic activity of granulocytes and slightly decreased respiratory burst. No significant differences in phagocytosis of monocytes were recorded. The percentage of CD3+, CD3+CD4+, CD3+CD8+, and CD3-CD19+ cell subsets in spleen, thymus, and lymph nodes did not differ between exposed and control animals. No changes in hematological parameters were found between the CuO NP exposed and control groups. The overall antioxidant protection status of the organism was expressed by evaluation of GSH and GSSG concentrations in blood samples. The experimental group exposed to CuO NPs showed a significant decrease in GSH concentration in comparison to the control group. In summary, our results indicate that sub-chronic inhalation of CuO NPs can cause undesired modulation of the immune response. Stimulation of adaptive immunity was indicated by activation of proliferation and secretion functions of lymphocytes. CuO NPs elicited pro-activation state of Th1 and Th2 lymphocytes in exposed mice. Innate immunity was affected by impaired phagocytic activity of granulocytes. Reduced glutathione was significantly decreased in mice exposed to CuO NPs.

2022

Coordinating and integrating UV observations in Svalbard.

Hansen, G.; Svendby, T.; Petkov, B.; Vitale, V.; Sobolewski, P.; Elster, J.; Laska, K.

2017

Coordinated lidar observations of Saharan dust over Europe in the frame of EARLINET-ASOS project during CALIPSO overpasses: a strong dust case study analysis with modeling support. NILU F

Papayannis A.; Amiridis, V.; Mona, L.; Mamouri, R.E.; Apituley, A.; Alados- Arboledas, L.; Balis, D.; Chaikovski, A.; De Tomasi, F.; Grigorov, I.; Gustafsson, O.; Linne, H.; Mattis, I.; Mitev, V.; Molero, F.; Müller, D.; Nicolae, D.; Pérez, C.; Pietruczuk, A.; Putaud, J.P.; Ravetta, F.; Rizi, V.; Schnell, F.; Sicard, M.; Simeonov, V.; Stebel, K.; Trickl, T.; D'Amico, G.; Pappalardo, G.; Wang, X.

2009

Coordinated lidar observations of Saharan dust over Europe in the frame of EARLINET-ASOS project during CALIPSO overpasses: a strong dust case study analysis with modeling support. Proceedings of the SPIE, 7479

Papayannis A.; Amiridis, V.; Mona, L.; Mamouri, R.E.; Apituley, A.; Alados- Arboledas, L.; Balis, D.; Chaikovski, A.; De Tomasi, F.; Grigorov, I.; Gustafsson, O.; Linne, H.; Mattis, I.; Mitev, V.; Molero, F.; Müller, D.; Nicolae, D.; Pérez, C.; Pietruczuk, A.; Putaud, J.P.; Ravetta, F.; Rizi, V.; Schnell, F.; Sicard, M.; Simeonov, V.; Stebel, K.; Trickl, T.; D'Amico, G.; Pappalardo, G.; Wang, X.

2009

Coordinated ground-based validation of ENVISAT atmospheric chemistry with NDSC network data: Commissioning phase report. ESA-SP531

Lambert, J.C.; Soebijanta, V.; Orsolini, Y.; Andersen, S.; Van, A. B.; Burrows, J.; Calisesi, Y.; Cambridge, C.; Claude, H.; De Backer-Barilly, M.-R.; de La Noë, J.; De Maziere, M.; Dorokohov, V.; Vik, A. F.; Godin-Beekmann, S.; Goutail, F.; Hansen, G.; Hochschild, G.; Høiskar, B. A.; Johnston, P.; Kämpfer, N.; Kreher, K.; Kyrö, E.; Leveau, J.; Mäder, J.; Milinevski, G.; Pommerau, J.-P.; Quinn, P.; Raffalski, U.; Rihter, A.; Roscoe, H.; Shanklin, J.; Staehlin, J.; Stebel, K.; Stubi, R.; Suortti, T.; Tørnkvist, K.; Van Roozendael, M.; Vaughan, G.; Wittrock, F.

2003

Coordinated European Particulate Matter Emission Inventory Program (CEPMEIP).

Visschedijk, A.; Pacyna, J.; Pulles, T.; Zandveld, P.; Denier van der Gon, H.

2007

Coordinated approach to improved environmental genotoxicity testing: EceGenoTox.

Shaposhnikov, S.; Brunborg, G.; Collins, A.R.; Dusinska, M.; Azqueta, A.; Fjellsbø, L.

2010

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